Why? 3/4 people do it on their own and then there's the yo-yo effect
- With Yo-Yo diets you end up with less lean muscle each time, lowering your resting metabolism
Our metabolism is broken down to:
- Resting Metabolic Rate (60-65% of your daily Energy expended)
- Thermogenic effect of food (~10% of kcal)
- Actively burning calories (exercise)
Clearly, if I were to put my money on the biggest contributor to weight management, I’d go with RMR. The goal here is to increase lean body mass (stimulate muscle).
95% of Diets DON'T WORK
Fat mobilization depends on hormone-sensitive lipase. Insulin blocks insulin-sensitive lipase in adipocytes (fat cells). In other words, fat goes in and doesn’t come out.
We may overeat because our fat cells are dysregulated where they are accumulating energy and we have no way of accessing it because insulin is chronically high. With this reasoning, we can starve an obese person but we won’t address or fix the cause.
“So what do I do?”
- Schedule a consulation with our team and we can establish a game plan!
Insulin & fat cell dysregulation
Fructose & Obesity
Fructose may arguably be the biggest contributor to obesity. Here we’ll highlight how the monosaccarride does so much damage:
Fructose is metabolized the same as ethanol by our liver, but it does not cross the blood-barrier, thus is not metabolized by the brain. It has been given the term: “alcohol, without the buzz.” Because it does not cross the blood-brain barrier it does not give us the same self-limiting dose of alcohol (dropping to the floor), contributing to why it is not being considered an ACUTE toxin by FDA (US) standards.
Fructose is converted to fat, especially the saturated fatty acid palmitate, increasing LDL
Note: fructose is stored as fat, not glycogen as glucose is
This fat accumulates in the liver and insulin resistance and metabolic syndrome follows
Fructose is a “chronic poison” because of these pathways:
- The production of citrate converts to acetyl CoA, leading to LDL buildup, contributing to obesity, leading to dyslipidemia (unhealthy amounts of fat in our blood)
- Production of the cytokine JNK-1creates inflammation
- De Nova lipogenesis--leading to non-alcoholic fatty liver (steatosis)
- It Inhibits glucose uptake in skeletal muscle, leading to insulin resistance
- Insulin blocks leptin from reaching the brain, so we experience hunger
- Uric acid (bi-product) buildup inhibits nitric oxide, leading to Hypertension
- Oxidative stress that accelerates kidney disease, causing microvascular lesions